Taking into account its detailed morphology, an attempt has been made
to explain a tendon's ability to transmit force between its muscular o
rigin and skeletal insertion. This ability depends upon the aggregatio
n and binding capacity of the molecular elements of type I collagen an
d is known to decrease with advancing age. The internal structure of a
tendon is modified both at the muscle-tendon junction and near its at
tachment to bone. The very low elasticity of tendinous tissue (about 3
% to 8%) is due partly to the texture (pseudoelasticity) and partly to
the molecular composition. The so-called degenerative changes in a te
ndon begin with deterioration of the internal vascular network and are
obviously - so far as its failing capacity is concerned - both initia
ted and accelerated by the degree of overloading.