The propagation of small cracks in fatigue has received considerable a
ttention over the past decade. Microstructurally and even mechanically
small cracks have been shown to consistently exhibit higher crack gro
wth rates than predicted using standard threshold and Paris growth law
concepts, based on linear elastic fracture mechanics (LEFM) applied t
o mechanically long cracks. This has been commonly attributed to sever
al factors, including the influence of microstructure, the breakdown o
f LEFM parameters for representation of the crack tip field, and the t
ransient development of plasticity induced closure towards some steady
-state value associated with long crack behavior. Other mechanisms rel
ated to microstructural effects such as roughness-induced closure and
crack face bridging/interference are also potential contributors. Quan
titative attempts to explain the fatigue propagation of small cracks i
n terms of plasticity-induced closure, along with adoption of an addit
ional component of the driving force (e.g. crack tip opening displacem
ent) to reflect the contribution of cyclic plastic strain have met wit
h some success in correlation of the so-called ''anomalous'' propagati
on behavior, including crack deceleration and acceleration transients.
However, these models rely on the adoption of highly idealized assump
tions regarding the self-similarity of crack growth, neglect of local
anisotropy and heterogeneity associated with microstructure, etc.; in
spite of these compromises, they still involve a considerable degree o
f complexity. Here, we adopt the viewpoint that multiple, microstructu
re interactions and closure effects may simultaneously influence the p
ropagation of small cracks; moreover, driving force parameters based o
n self-similar crack growth arguments of elastic-plastic fracture mech
anics (EPFM) for mechanically long cracks, such as the cyclic J-integr
al or crack tip opening displacement, may apply in principle, but not
rigorously, as the driving force for small cracks. As an engineering a
pproach, we consider a recent extension of the multiaxial microcrack p
ropagation model first proposed by McDowell and Berard[1,2] for the gr
owth of microstructurally small and mechanically small fatigue cracks[
3] in multiaxial fatigue. Integrated between initial and final crack l
engths, the model is fully consistent with standard strain-life laws o
f fatigue crack initiation mechanics under various states of stress, [
1,2] and therefore bridges the mechanics of classical initiation and L
EFM/EPFM to some extent. The existence of a fatigue limit (nonpropagat
ing crack limit) is neglected in this particular work. It is shown for
uniaxial loading of both 1045 steel and Inconel 718 that the model is
able to describe, to first order, the anomalous high propagation rate
s of small cracks and convergence with long crack da/dN-Delta K data a
s the crack transitions from small to mechanically long scales. The li
mits of validity of engineering schemes based on decomposition of tota
l fatigue life into ''initiation'' and propagation phases that rely on
strainlife and long crack propagation laws are discussed[4]. Moreover
, it is shown that the model essentially reflects a closure transient
in the context of a cyclic J-integral approach, similar to the EPFM pl
asticity-induced closure modelling concepts set forth by Newman[5] and
McClung et al. [6] for small cracks in fatigue. However, the present
model implicitly reflects multiple forms of crack tip shielding effect
s, not just plasticity-induced closure. Finally, the model is shown to
provide realistic treatment of cumulative damage in two level loading
sequences, as reflected by comparison with the damage curve approach
of Manson and Halford[7]. Copyright (C) 1996 Elsevier Science Ltd