PAIN COPING MECHANISMS IN FIBROMYALGIA - RELATIONSHIP TO PAIN AND FUNCTIONAL OUTCOMES

Citation
Pm. Nicassio et al., PAIN COPING MECHANISMS IN FIBROMYALGIA - RELATIONSHIP TO PAIN AND FUNCTIONAL OUTCOMES, Journal of rheumatology, 22(8), 1995, pp. 1552-1558
Citations number
34
Categorie Soggetti
Rheumatology
Journal title
ISSN journal
0315162X
Volume
22
Issue
8
Year of publication
1995
Pages
1552 - 1558
Database
ISI
SICI code
0315-162X(1995)22:8<1552:PCMIF->2.0.ZU;2-F
Abstract
Objective. To evaluate the factor structure of the Coping Strategies Q uestionnaire (CSQ) in patients with fibromyalgia (FM) and to compare t he factors derived from this measure, along with the active and passiv e pain coping scales of the Pain Management Inventory (PMI) in predict ing pain, depression, quality of well being (QWB), and pain behavior c oncurrently and over time. Methods. One hundred twenty-two patients wi th FM were recruited from medical clinics, the community, and support groups. Eligible patients completed a battery of self-report measures of pain and psychosocial functioning at baseline assessment before ran dom assignment to a clinical trial. A subset of 69 patients who comple ted the clinical trial were readministered the same battery 3 mo later . Data were analyzed within the baseline period, and from the baseline period to posttreatment to evaluate the predictive effects of coping strategies on clinical outcomes. Results. Principal components analysi s of the CSQ revealed Coping Attempts (CA) and Pain Control and Ration al Thinking (PCRT) factors, which have been found in other patient pop ulations with chronic pain. Hierarchical multiple regression analyses revealed that high active coping and low PCRT contributed to higher co ncurrent pain, while low active coping and high passive coping were re lated to greater concurrent depression and pain behavior, respectively . Controlling for baseline scores on criterion measures, longitudinal multiple regression analyses demonstrated that high active coping and low PCRT scores contributed to greater pain, greater depression, and l ower QWB at posttreatment, while low PCRT alone predicted greater pain behavior. Conclusion. The results show the import of the pain coping construct in FM and highlight the negative contribution of low perceiv ed control over pain and high active coping to a range of pain outcome s. The findings on low perceived control converge with data on other c hronic pain populations, while the role of active coping appears to be detrimental in FM, in contrast to its positive effects in patients wi th rheumatoid arthritis.