To date, pulmonary oedema in breath hold divers has only been reported
after dives below 50 meters, hypoxaemic syncope being the most common
risk We recently observed a 35-year-old well-trained breath hold dive
r who was unable to achieve deep inspiration during a high-level compe
tition, After two hours of repeated dives to a depth of 25 meters for
approximately 2-minute periods with intermittent recovery the patient
developed cough and haemoptysis. The chest X-ray revealed lung images
suggestive of intra-alveolar haemorrhage, The patient had taken 1 g of
aspirin per os for three days prior to diving, Symptoms subsided spon
taneously in 48 hours and one month later all haematology tests were n
ormal except for minimal alteration of platelet aggregation. Pulmonary
oedema in breath hold divers is usually atributed to blood shift to t
he pulmonary circulation related to the lowered intra-thoracic pressur
e, In our case, oedema was secondary to intra-alveolar haemorrhage fav
oured by aspirin which should be avoided before breath hold diving.