A 63-year-old woman admitted with 2:1 infranodal atrioventricular bloc
k subsequently developed ventricular dysfunction incident to septic sy
ndrome. Concomitant changes included an abnormally prolonged QTc inter
val (600 ms) and the occurrence of torsade de pointes. Restoration of
a normal QTc interval and cessation of torsade de pointes was coincide
nt with return of normal ventricular function and remission of sepsis.
This report supports the view that sepsis-induced cardiomyopathy is a
nother cause of the long QT syndrome.