Depressed immunity in uremic patients increases by ten the risk of tub
erculosis. In such patients, 40% of tuberculosis manifestations are ex
trapulmonary, and peritoneum is involved in about 6% of the cases. Sev
enteen cases of peritoneal tuberculosis have been so far reported in C
APD patients, and we add a new case. The prognosis of the disease is s
evere since 8 patients died. Three deaths out of 8 are directly linked
to tuberculosis. Indeed, peritoneal tuberculosis diagnosis is hard an
d often late, at least for two main reasons: at first, it can be diffi
cult to exclude the other causes of lymphocytic peritonitis (viral, fu
ngal, bacterial, etc..), secondly, growth of mycobacteria in dialysate
effluent cultures is late and inconstant. Omental biopsy in lymphocyt
ic peritonitis of unknown origin could be of great value for an early
diagnosis. Despite the adaptation of antituberculous drugs doses, side
-effects are not so rare: optical neuritis and liver toxicity in our c
ase. In spite of ultrafiltration loss, stopping CAPD is not always nec
essary, as in the reported case.