ABSENCE OF ENHANCEMENT OF SPONTANEOUS ECHOCARDIOGRAPHIC CONTRAST BY THROMBOCYTOSIS IN A PATIENT WITH LEFT-VENTRICULAR ANEURYSM, PRIMARY THROMBOCYTHEMIA, AND VONRECKLINGHAUSEN NEUROFIBROMATOSIS - A CASE-REPORT

A thirty-eight-year-old man with primary thrombocythemia, von Reckling hausen neurofibromatosis, and myocardial-infarction-related left ventr icular aneurysm with spontaneous echocardiographic contrast was follow ed up, suggesting that: 1. Neurofibromatosis may promote silent myocar dial infarction or ischemia. Whether involvement of cardiac sensory ne rves is a possible underlying mechanism remains nevertheless uncertain . 2. Platelets, whose role in the genesis of spontaneous echocardiogra phic contrast has been advocated, are probably not involved in this ph enomenon, even in large numbers.