Rebound caloric nystagmus (RCN), a distinctly different phenomenon fro
m secondary phase nystagmus (SPN), was serendipitously discovered duri
ng the course of routine neurotological evaluations. RCN occurred when
the horizontal semicircular canal was brought into a truly horizontal
plane after the primary phase nystagmus (PPN) had ended. The maximum
slow-phase velocities (SPV(Max)) of both PPN and RCN were almost ident
ical. The pathological lesion was an Arnold-Chiari malformation (ACM).
Two other patients with posterior fossa lesions also showed RCN. To d
etermine if RCN is an unrecognized normal phenomenon, 11 healthy subje
cts were tested. The average SPV of RCN in normals was 0.7 degrees per
second. Such a small value of RCN is probably of no significance. It
therefore appears that RCN is a sign of posterior fossa neuroaxial les
ions.