Five of seven patients with chronic nonfluent aphasia and hemiparesis
due to a focal ischemic infarction developed painful hemidystonia duri
ng treatment with a high dose of bromocriptine. All seven patients had
cortical damage, but four also had basal ganglia and one thalamic inv
olvement. While lesion location did not differ between dystonic and no
ndystonic patients, the dystonic patients had more weakness than those
without dystonia.