In the last 5 years the authors had 10 patients with Ogilvie's syndrom
e under observation. The frequency of the development of the syndrome
among all patients who were in the department in this period of time w
as 0.09%, those who underwent operation accounted for 0.1%. Mortality
was 60%. Seven patients were treated by operation. In all patients the
syndrome developed in a severe internal disease of an operative inter
vention. The authors encountered the following clinical diagnostic sig
ns in these patients: moistened tongue, mild tachycardia, absence of h
ypertrophy of the antestinal wall, and no fecal masses found in the lu
men of the large intestine on operation.