Ab. Brill et al., RADON UPDATE - FACTS CONCERNING ENVIRONMENTAL RADON - LEVELS, MITIGATION STRATEGIES, DOSIMETRY, EFFECTS AND GUIDELINES, The Journal of nuclear medicine, 35(2), 1994, pp. 368-385
The risk from environmental radon levels is not higher now than in the
past, when residential exposures were not considered to be a signific
ant health hazard. The majority of the radon dose is not from radon it
self, but from short-lived alpha-emitting radon daughters, most notabl
y Po-218 (T-1/2 3 min) and Po-214 (T-1/2 0.164 msec) along with beta p
articles from Bi-214 (T-1/2 19.7 min). Radon gas can penetrate homes f
rom many sources and in various fashions. Measuring radon in homes is
simple and relatively inexpensive and may be accomplished in a variety
of ways. Although it is not possible to radon-proof a house, it is po
ssible to reduce the level. In high radon areas, if the average level
is higher than 4-8 pCi/liter (NCRP recommended level is 8 pCi/liter; E
PA recommended level is 4 pCi/liter), appropriate action is advised. T
he shape of the dose response curves for miners exposed to alpha-emitt
ing particles in the workplace is consistent with current biologic kno
wledge. It is linear in the low dose range and saturates in the high d
ose range. No detectable increase in lung cancer frequency is seen in
the lowest exposed miners (those with exposures <120 WLM, the relevant
dose interval for most homes). Evidence for a health effect from rado
n exposure is based on data from animal studies and epidemiologic stud
ies of mines. Extensive radiobiologic data predict a linear dose-respo
nse curve in the low dose region due to poor biological repair mechani
sms for the high density of ionizing events that alpha particles creat
e. However, no compelling evidence for increased cancer risks has yet
been demonstrated from ''acceptable'' levels (<4-8 pCi/liter).