The etiology of the endocrine orbitopathy is still unknown and thereby
their treatment remains symptomatic. The surgical decompression is ac
hieved in resecting lateral wall, roof and partly bottom of the orbit.
We treated 23 eyes in 14 patients and in only one patient the decompr
ession obtained whis this method was insufficient. This patient underw
ent additionally transantral transethmoidal decompression. One patient
had a complete loss of vision after surgery probably du to direct les
ion of the optic nerve. The decompression of the orbit is indicated in
fast progressive endocrine orbitopathies with impairement of the visu
al fonction.