The hemodynamic consequences of ventricular tachycardias are caused by
cardiac and peripheral reactions. As a result, cardiac output and art
erial pressure decrease. Even if the decrease is related to the tachyc
ardia rate and left ventricular function, clinical symptoms do not in
each case correlate with the degree of hemodynamic compromise. The exp
lanation for this phenomenon is the different cardiac and peripheral r
eaction of patients to an immediate rise in heart rate. In this regard
, it is questionable if pressure monitoring is superior to heart rate
monitoring as a sensor for implantable antitachycardia devices.