Significant insight has been gained in the past year into the roles of
salicylic acid (SA) in plant-pathogen interactions. The ability to ac
cumulate SA has been shown to be essential for systemic acquired resis
tance in tobacco plants. Further experiments have shown that SA is app
arently not a systemic, vascular-mobile signal, but rather is required
for signal transduction at the local level. Its mode of action may in
clude inhibition of catalase activity, leading to increased levels of
hydrogen peroxide.