TRANS-ETHMOIDAL OPTIC-NERVE DECOMPRESSION

Citation
Da. Tandon et al., TRANS-ETHMOIDAL OPTIC-NERVE DECOMPRESSION, Clinical otolaryngology and allied sciences, 19(2), 1994, pp. 98-104
Citations number
21
Categorie Soggetti
Otorhinolaryngology
ISSN journal
03077772
Volume
19
Issue
2
Year of publication
1994
Pages
98 - 104
Database
ISI
SICI code
0307-7772(1994)19:2<98:TOD>2.0.ZU;2-W
Abstract
Blunt head injury frequently results in visual impairment, the optimal treatment of which is still debated. Over a 5-year-period (1987-1991) 111 patients with indirect optic nerve injury resulting from closed h ead trauma have been treated. In each case loss of vision was the only neurological deficit. In group A, 66 patients were initially treated with large doses of prednisolone (80 mg/day) for 3 weeks. Twenty-seven patients improved on steroids alone. In the remaining 39 patients in whom either unsatisfactory or no improvement occurred a transethmoidal optic nerve decompression was performed. Twenty-two patients in the l atter group improved, thus yielding an overall improvement rate of 74. 2% in group A. Group B (control), in which 45 patients were treated wi th prednisolone only (80 mg/day for 3 weeks), had an overall improveme nt rate of 51% (23 patients). The study reveals that while nearly half of such patients can improve on steroids alone, optic nerve decompres sion significantly improves recovery rates in patients where conservat ive treatment is unsatisfactory (P < 0.05). Total loss of vision not r esponding to steroids, absence of waveform on visual evoked response, and presence of-an optic canal fracture indicate a poor prognosis.