Blunt head injury frequently results in visual impairment, the optimal
treatment of which is still debated. Over a 5-year-period (1987-1991)
111 patients with indirect optic nerve injury resulting from closed h
ead trauma have been treated. In each case loss of vision was the only
neurological deficit. In group A, 66 patients were initially treated
with large doses of prednisolone (80 mg/day) for 3 weeks. Twenty-seven
patients improved on steroids alone. In the remaining 39 patients in
whom either unsatisfactory or no improvement occurred a transethmoidal
optic nerve decompression was performed. Twenty-two patients in the l
atter group improved, thus yielding an overall improvement rate of 74.
2% in group A. Group B (control), in which 45 patients were treated wi
th prednisolone only (80 mg/day for 3 weeks), had an overall improveme
nt rate of 51% (23 patients). The study reveals that while nearly half
of such patients can improve on steroids alone, optic nerve decompres
sion significantly improves recovery rates in patients where conservat
ive treatment is unsatisfactory (P < 0.05). Total loss of vision not r
esponding to steroids, absence of waveform on visual evoked response,
and presence of-an optic canal fracture indicate a poor prognosis.