In F2 progeny, derived from F1 hybrids, shoot growth of seedlings, mea
sured 4 d after germination, distinguished susceptible (S) and resista
nt (R) phenotypes. Chi-square values indicated that the F2 data fit a
3:1 (S:R) ratio for both populations and all trifluralin concentration
s in which S and R phenotypes could be differentiated. Results indicat
e that trifluralin resistance in these green foxtail populations is co
ntrolled by a single, nuclear recessive gene. This study is the first
to demonstrate recessive gene control of herbicide resistance in a wee
d species. The highly selfed nature and prolific seed production of gr
een foxtail may have facilitated evolution of the recessive trait.