BACKGROUND: Atrioventricular nodal reentrant tachycardia (AVNRT) is th
e most common form of regular narrow complex tachycardia. It is due to
dual atrioventricular nodal conduction over two pathways with differe
nt electrophysiological properties. The first pathway ('fast' pathway)
conducts faster but has longer refractory period than the second path
way ('slow' pathway). OBJECTIVES: To review AVNRT. Clinically, AVNRT p
atients usually have palpitations in their neck during attacks. On the
surface electrocardiogram, the diagnosis is suggested by the absence
of P waves during tachycardia or very discrete P waves immediately aft
er the QRS or an rSr' pattern in lead VI. Electrophysiologically, it c
an be reproducibly initiated or terminated by cardiac pacing. The reen
trant circuit is limited to the atrioventricular node and a small amou
nt of perinodal atrial tissue. Acute termination of tachycardia can be
achieved by vagal manoeuvres or drugs. Adenosine compounds are excell
ent drugs, as are calcium channel blockers, for acute termination of t
he arrhythmia. If chronic therapy is indicated, digitaiis, calcium blo
ckers and beta-blockers are effective and simple initial options. Cath
eter ablation, especially using radiofrequency energy, antitachycardia
pacing and surgery are therapeutic alternatives for the resistant pat
ient. CONCLUSION: Because of its high success rate and low incidence o
f complications, radiofrequency ablation is becoming the therapy of fi
rst choice for the treatment of AVNRT.