Faecal incontinence is a disabling condition caused by: (1) sphincter
damage caused by childbirth, anorectal surgery, trauma, fistulae and a
bcesses; (2) pudendal neuropathy (''idiopathic faecal incontinence'')
caused by stretch injury by long-standing constipation or prolonged la
bor; (3) diminished rectal compliance in proctitis, low anterior resec
tion or small pouches; (4) faecal impaction causing paradoxal diarrhoe
a; (5) neurological disease involving the pelvic floor and or the cent
ral nervous system; (6) diarrhoea. Often several factors play a role i
n a patient. A medical history and physical examination will generally
provide a reasonable diagnosis. Anorectal function tests can show one
or more abnormalities. Anal manometry can show low sphincter pressure
s; rectal compliance can show a small Tectal volume; anal mucosal sens
itivity measurement can show a high threshold and neurophysiological t
ests can demonstrate diminished muscle activity and a delayed pudendal
nerve motor latency. Anal endosonography and defaecography have a dir
ect clinical impact. Anal endosonography is a promising diagnostic too
l demonstrating sphincter defects, even those not previously suspected
. A sphincter defect demonstrated by anal endosonography provides a so
lid basis for a sphincter repair. Defaecography can reveal an intussus
ception, which is an indication for performing a rectopexy in the inco
ntinent patient. A suggested work-up of the incontinent patient is giv
en in a table. Besides the classic surgical treatments such as sphinct
er repair, rectopexy and post-anal repair new (surgical) options have
been tried. The most promising new therapy seems the dynamic gracilis
repair.