In the last 14 years we have encountered 16 patients with spontaneous
intracranial dissections of the vertebrobasilar (VB) system (mean age
was 46 years and male/female ratio was 10/6). VB dissections presented
in eight cases with subarachnoid bleeding and in eight with brainstem
, cerebellar, or cerebral ischemia. Three patients had multiple dissec
tions. Ten dissections occurred in the vertebral artery (two extended
to the basilar artery), three in the posterior cerebral artery (one bi
lateral), two in the basilar artery, and one in the posterior inferior
cerebellar artery. The angiographic configuration included ''string''
sign, ''pearl and string'' sign, fusiform dilation, and double lumen.
The following angiographic evolution (available in 11 cases) was that
of complete healing (three cases), partial resolution (five cases), p
rogression (one case), and unimproving (two cases). Three patients die
d (two due to recurrent subarachnoid hemorrhage): two patients were ex
plored surgically (one had further intravascular therapeutic embolizat
ion), and the rest were treated conservatively. Not all dissecting ane
urysms fared in the same manner, depending either on the location in t
he VB circulation or on the variable vascular configuration: treatment
should be fitted to the timing of diagnosis.