S. Albrecht et al., PATHOANATOMIC FINDINGS IN RADIOHUMERAL EPICONDYLOPATHY - A COMBINED ANATOMIC AND ELECTROMYOGRAPHIC STUDY, Archives of orthopaedic and trauma surgery, 116(3), 1997, pp. 157-163
Several authors believe that a compression syndrome of the radial nerv
e or its muscle branches is responsible for the clinical picture of ra
diohumeral epicondylopathy. Various structural and functional stenoses
have been discussed as possible causes. We performed systematic elect
romyographies (EMGs) on the extensors subdividing from the radial epic
ondyle and found significant changes (P < 0.05) in 27/51 patients rega
rding latency, velocity of nerve conduction and rate of polyphasic pot
entials. Especially affected were the extensor carpi radialis brevis a
nd the extensor digitorum muscle. In order to clarify causal anatomic
correlations, we performed a longitudinal and cross-sectional study on
a total of 40 arms from cadavers. We found constant variations from t
he topographic anatomy published in the standard literature which corr
esponded to the EMG results in the area between the epicondyle and pla
ce of entry into the supinator muscle. In addition, we observed a regu
lary occurring ulnar deviation from the distal part of the extensor ca
rpi radials brevis origin which protrudes over the plane of insertion
of the joint extensor tendon aponeurosis and forms in most cases the a
rcade of Frohse. Because the deep radial branch and its parallel muscu
lar branches cross this part at an obtuse angle, we think that dynamic
pressure on a nerve without structural influences is the pathoanatomi
c result of this heterogeneously interpreted clinical picture.