The genetic and biological nature of a deficiency in axonal regenerati
on in C57BL/6J mice was investigated. From analysis of recombinant inb
red strains, the genetic basis for the deficient regeneration behaviou
rs of C57BL/6J and A/J mice is deduced to involve multiple loci. The d
efect in axonal regeneration in C57BL/6J mice appears to be a delay ra
ther than permanant impairment and appears to involve sensory more tha
n motor axons.