Simulations of squall lines, using nonhydrostatic convection-resolving
models, have been limited to two dimensions or three dimensions with
the assumption of along-line periodicity. The authors present 3D nonhy
drostatic convection-resolving simulations, produced using an adaptive
grid model, where the lines are finite in length and the restriction
to along-line periodicity is removed. The base state for the simulatio
ns is characterized by weak, shallow shear and high convective availab
le potential energy (CAPE), an environment in which long-lived midlati
tude mesoscale convective systems (MCSs) are observed. The simulated s
ystems bear strong resemblance to many observed systems, suggesting th
at large-scale forcing, absent in the horizontally homogeneous environ
ment, is not needed to produce many of the distinguishing features of
midlatitude MCSs. In simulations without Coriolis forcing, the presenc
e of line ends leads to mature symmetric systems characterized by a ce
ntral region of strong convection, trailing flanks of weaker convectio
n, and a strong, centrally focused rear inflow. Simulations that inclu
de Coriolis forcing lead to asymmetric systems with significant system
growth and migration to the right (south) of the original system cent
erline. In both cases the evolution of the leading-line convection is
primarily controlled by the surface cold pool expansion, with Coriolis
forcing promoting rightward system propagation. In the Coriolis simul
ation, a midlevel mesoscale convective vortex (MCV) forms in the north
, to the rear of the convection, while the outflow region aloft is str
ongly anticyclonic. The northern location of the MCV is coincident wit
h and influenced by a northward bias in the positive buoyancy anomaly
aloft. Midlevel vertical vorticity generation by tilting of horizontal
vorticity, both ambient and baro-clinically generated, is observed in
both the Coriolis and no-Coriolis simulations. On larger scales, the
convergence of Coriolis rotation generates significant vorticity and i
s crucial to the formation of the MCV.