BASAL GANGLIA AND THALAMIC CALCIFICATION FOLLOWING INTRAUTERINE INTRAVASCULAR TRANSFUSION

In recent years intrauterine intravascular transfusion has become the method of choice for the treatment of severe fetal erythroblastosis. D espite the association of the procedure with fetal complications such as bradycardia and overtransfusion, later neonatal neurological conseq uences have been only rarely reported. A case is described of a neonat e who underwent repealed intrauterine intravascular transfusion for tr eatment of erythroblastosis and who later developed calcification in t he thalamus and the basal ganglia. The possible pathogenetic mechanism s are discussed.