T-WAVE CHANGES CONSISTENT WITH EPICARDIAL INVOLVEMENT IN ACUTE MYOCARDIAL-INFARCTION - OBSERVATIONS IN PATIENTS WITH A POSTINFARCTION PERICARDIAL-EFFUSION WITHOUT CLINICALLY RECOGNIZED POSTINFARCTION PERICARDITIS
Pb. Oliva et al., T-WAVE CHANGES CONSISTENT WITH EPICARDIAL INVOLVEMENT IN ACUTE MYOCARDIAL-INFARCTION - OBSERVATIONS IN PATIENTS WITH A POSTINFARCTION PERICARDIAL-EFFUSION WITHOUT CLINICALLY RECOGNIZED POSTINFARCTION PERICARDITIS, Journal of the American College of Cardiology, 24(4), 1994, pp. 1073-1077
Objectives. This study was designed to evaluate the presence or absenc
e of atypical T wave evolution in patients with a postinfarction peric
ardial effusion but without clinically recognized postinfarction peric
arditis. A second purpose was to evaluate the frequency of atypical T
wave evolution in a previous study of postinfarction pericarditis. Bac
kground. Electrocardiographic (ECG) criteria involving the evolution o
f the T wave after an acute myocardial infarction were recently descri
bed in patients with regional postinfarction pericarditis. Atypical T
wave evolution was found to have a sensitivity of 100% and a specifici
ty of 77% for clinically recognized regional postinfarction pericardit
is with or without a pericardial effusion. Methods. The hospital recor
ds and serial ECGs of 20 patients with clinically recognized postinfar
ction pericarditis (Group I) were reviewed. The records and serial ECG
s of 20 additional patients with a postinfarction pericardial effusion
without clinically recognized postinfarction pericarditis (Group II)
were also examined. The type of postinfarction T wave pattern, typical
or atypical, was recorded in both groups. Results. All 20 patients in
Group I had atypical T wave evolution. Among the 20 patients in Group
II, every patient also had atypical T wave evolution. Fifteen percent
of all 40 patients with atypical T wave evolution had a non-Q wave in
farction with definite or inferred postinfarction pericarditis. Conclu
sions. The high sensitivity of atypical T wave evolution in diagnosing
regional postinfarction pericarditis was confirmed. However, similar
T wave alterations were also observed when a postinfarction pericardia
l effusion existed in the absence of clinically recognized pericarditi
s. Fifteen percent of patients with atypical T wave evolution had a no
n-Q wave infarction with definite or inferred pericardial involvement.
Thus, the presence of atypical T wave evolution may be a more sensiti
ve indicator of a transmural infarction than the development of a Q wa
ve.