A total of 20 patients (16 males and 4 females) with repetitive nonpar
oxysmal ventricular tachycardia (RNVT) underwent Holter monitoring of
ECG, bicycle exercise test, intracardiac electrophysiological measurem
ents. The findings evidence for a principal difference of this tachyca
rdia form from paroxysmal ventricular tachycardia by trigger mechanism
. All the tests performed showed rhythm-dependence of RNVT, tachycardi
a cannot be initiated by electrocardiostimulation, but can be stopped
after intravenous introduction of finoptin. It is evident that this fo
rm of cardiac arrhythmia is due to the mechanism of pathological ectop
ic automatism represented, most likely, by trigger activity, but not r
e-entry, as it is in the majority of patients with paroxysmal ventricu
lar tachycardia.