B. Benjamin et al., ESOPHAGEAL STRICTURE FOLLOWING ESOPHAGEAL ATRESIA REPAIR - ENDOSCOPICASSESSMENT AND DILATION, The Annals of otology, rhinology & laryngology, 102(5), 1993, pp. 332-336
A retrospective review from 1981 to 1986 was made of 51 neonates who w
ere treated for congenital esophageal atresia at the Royal Alexandra H
ospital for Children, Sydney, Australia. Thirty of the 51 had primary
repair and 15 (50%) developed esophageal stricture requiring one or mo
re dilations. Stricture dilations were performed with open-tube rigid
esophagoscopes, rigid telescopes, and Jackson dilators. The number of
dilations performed until the patient was asymptomatic ranged from 1 t
o 12 (mean 4.5). The minimal follow-up time was 4 years. There was one
esophageal perforation with full recovery following conservative mana
gement. One child died of septicemia caused by continued aspiration of
unknown cause. A surprising finding, contrary to other reports, was t
hat stricture formation did not appear to be related to the size of th
e ''gap'' found at the time of esophageal repair. Gastroesophageal ref
lux, however, was an important factor. We conclude that esophageal str
icture following repair of tracheoesophageal atresia can be satisfacto
rily and relatively safely managed by direct vision antegrade endoscop
ic dilation using graduated dilators, although treatment may be prolon
ged and repeated. New esophagoscopes and dilators, in graduated sizes
and designed for pediatric use, are recommended. The technique of esop
hageal dilation in infants is described in detail. Pediatric laryngobr
onchoesophagologists will become more involved in the care of infants
born with esophageal atresia with or without tracheoesophageal fistula
.