Eight cases of diurnal bruxism (DB) secondary to long-term antidopamin
ergic drug exposure are reported. Five exhibited a grinding pattern, o
ne a clenching form, and two a mixed type. An odontological etiology w
as absent throughout. EMG recordings disclosed two distinct patterns o
f muscle activity, one with brief rhythmic, forceful contractions and
the other featuring sustained prolonged contractions. Surface EMG and
EEG monitoring during a 24-h period confirmed the absence of bruxism d
uring sleep. Several drug trials failed to provide relief. Our finding
s support DB as a focal tardive dystonia syndrome.