The etiology of rotator cuff disease is controversial, but is not alwa
ys secondary to cuff impingement between the humeral head and the cora
coacromial arch. Rotator cuff disease, and not impingement syndrome, m
ore accurately describes this process. The spectrum of rotator cuff di
sease begins with rotator cuff inflammation (Stage I) and progresses t
o tendinitis (Stage II) and partial or full thickness tears (Stage III
). Refractory Stage II and III disease without rotator cuff tears unde
rgoes subacromial bursectomy and acromioplasty. Partial thickness tear
s are debrided with an acromioplasty, bursectomy, and removal of infer
ior clavicular osteophytes. Full thickness tears are treated arthrosco
pically or open, depending on the tear size and the patient expectatio
ns. In general, tears from 0 to 5 cm in active patients undergo subacr
omial decompression and a mini-open repair. Low demand patients with 0
to 5 cm tears or tears greater than 5 cm are treated with subacromial
decompression and rotator cuff debridement.