Rf. Heary et al., NONSURGICAL TREATMENT OF COMPOUND DEPRESSED SKULL FRACTURES, The journal of trauma, injury, infection, and critical care, 35(3), 1993, pp. 441-447
Classical treatment of compound depressed skull fractures includes deb
ridement and closure of all scalp wounds to minimize the risk of delay
ed intracranial infection. In selected situations, a nonsurgical appro
ach may prove to be equally safe. Over a 33-month period, we evaluated
1075 patients for head trauma. There were 63 patients with non-missil
e, compound depressed skull fractures (9 of these 63 patients have bee
n excluded for deaths that occurred within 4 days of admission). Patie
nts with significant intracranial hematomas or dural violations over t
he convexity of the brain underwent formal surgical therapy. Surgical
therapy was performed on 28 patients (52%; age 33 +/- 3 years; ISS 24
+/- 2) and it consisted of craniotomy with debridement, elevation of d
epressed fragments, repair of dural tears, and evacuation of hematomas
. Nonsurgical treatment was used if there was no evidence of violation
of the dura mater and of significant intracranial hematoma. If the fo
llowing criteria were satisfied, then the nonsurgical approach was emp
loyed: no evidence of exposed brain or a cerebrospinal fluid leak, no-
pneumocephalus related to the fracture, no depressed fragments of bone
more than 1 cm below the inner table of the skull, and no gross wound
contamination. Nonsurgical therapy was used on 26 patients (48%; age
34 +/- 3 years; ISS 19 +/- 2) and it consisted of wound irrigation, de
bridement, and closure. In all 54 patients of both subsets of patients
, intravenous antibiotics were administered for 5 to 7 days. After two
additional days of observation, off antibiotics, the patients were di
scharged. There were no infectious complications related to the centra
l nervous system. Patients with open skull fractures who do not demons
trate evidence, either clinically or radiographically, of violation of
the dura mater or of a significant underlying brain injury may be tre
ated with local wound care and intravenous antibiotics without an incr
eased risk of a subsequent central nervous system infection.