This paper evaluates the uncertainties of an impact pathway analysis which
traces the fate of each pollutant or other burden, from the source to the r
eceptors, using dose-response functions to evaluate the damage. The express
ion for the total damage is shown to be largely multiplicative, even though
it involves a sum over receptors at different sites. This follows from con
servation of matter which implies that overprediction of the dispersion mod
el at one site is compensated by underprediction at another; the net error
of the total damage arises mostly from uncertainties in the rate at which t
he pollutant disappears from the environment. Since the central limit theor
em implies that the error distribution for multiplicative processes is like
ly to be approximately lognormal, one may be able to bypass the need for a
detailed and tedious Monte Carlo calculation. Typical error distributions a
re discussed for the factors in the expression for the total damage, in par
ticular those of two key parameters: the deposition velocity of atmospheric
dispersion models, and the value of statistical life; they are close to lo
gnormal. A lognormal distribution for the total damage appears plausible wh
enever the dose-response function is positive everywhere. As an illustratio
n, results for several types of air pollution damage are shown (health dama
ge due to particles and carcinogens, damage to buildings due to SO2, and cr
op losses due to O-3): the geometric standard deviation is in the range of
3 to 5. To the extent that the distribution of the result is lognormal, the
geometric mean equals the median and the geometric standard deviation has
a simple interpretation in terms of multiplicative confidence intervals aro
und the median. (C) 1999 Elsevier Science Ltd.