The aim of rehabilitation is to improve exercise capacity and, thereby, the
autonomy of patients with cardiac failure. For many years, these patients
were considered inapt to perform physical exercise and they are in the same
situation at the dawn of the year 2000 as patients with myocardial infarct
ion forty years ago.
The symptoms of cardiac failure (dyspnoea of effort and muscular fatigue) a
re not only the consequence of pulmonary hypertension and decreased muscula
r perfusion. Prolonged interruption of exercise and long stays in bed or in
a chair lead to anatomical and functional amyotrophy, which, in turns, inc
ites to further inactivity. Deconditioned respiratory muscles cannot tolera
te the increased load of hyperventilation. Neurohormonal changes cause vaso
constriction which reduces muscular perfusion.
Physical training can significantly improve these abnormalities, though it
does not seem to have a measurable effect on cardiac function; based on seg
mental work which enables performance of substantial efforts with a minimum
of haemodynamic changes, it provides a 20 to 30% gain in capacity, mainly
increasing the duration of submaximal exercise rather than maximum performa
nce. Muscular fatigue is the symptom which is the most improved.
Unfortunately the organisation, which is more difficult than in the post-in
farction period, and the generalisation of the practice of long-term, well
adapted physical training remains marginal although hundreds of thousands o
f patients could benefit; more than the inertia of the official instances c
oncerning anything related to cardiac rehabilitation, it is the lack of int
erest shown by cardiologists and the absence of flexible structures within
the health care organisation for elderly people which are responsible.