Following damage to primary visual cortex, some patients demonstrate a
limited ability to respond to stimuli they do not acknowledge seeing.
This residual vision, which has been referred to as ''blindsight,'' h
as been attributed to secondary visual pathways. We previously reporte
d an isolated island of blindsight in one patient and argued it was a
likely consequence of cortical sparing in V1. We now report an extensi
on of our initial findings. The data reveal a patchy distribution of r
esidual visual abilities in the absence of acknowledged awareness. Var
iable patterns of cortical sparing appear to be the most parsimonious
way to account for this outcome, suggesting that blindsight is general
ly mediated by the primary visual pathway.