M. Sandsund et al., EFFECT OF COLD-EXPOSURE (-15-DEGREES-C) AND SALBUTAMOL TREATMENT ON PHYSICAL PERFORMANCE IN ELITE NONASTHMATIC CROSS-COUNTRY SKIERS, European journal of applied physiology and occupational physiology, 77(4), 1998, pp. 297-304
The effects of whole-body exposure to ambient temperatures of -15 degr
ees C and 23 degrees C on selected performance-related physiological v
ariables were investigated in elite nonasthmatic cross-country skiers.
At an ambient temperature of -15 degrees C we also studied the effect
s of the selective beta(2)-adrenergic agonist Salbutamol (0.4 mg x 3)
which was administered 10 min before the exercise test. Eight male cro
ss-country skiers with known maximal oxygen uptakes ((V) over dotO(2ma
x)) of more than 70 ml . kg(-1) min(-1) participated in the study. Oxy
gen uptake ((V) over dotO(2)), heart rate (f(c)), blood lactate concen
tration ([La-](b)) and time to exhaustion were measured during control
led submaximal and maximal running on a treadmill in a climatic chambe
r. Lung function measured as forced expiratory volume in 1 s (FEV1) wa
s recorded immediately before the warm-up period and at the conclusion
of the exercise protocol. Submaximal (V) over dotO(2) and [La-](b) at
the two highest submaximal exercise intensities were significantly hi
gher at -15 degrees C than at 23 degrees C. Time to exhaustion was sig
nificantly shorter in the cold environment. However, no differences in
(V) over dotO(2max) or f(c) were observed. Our results would suggest
that exercise stress is higher at submaximal exercise intensities in a
cold environment and support the contention that aerobic capacity is
not altered by cold exposure. Furthermore, we found that after Salbuta
mol inhalation FEV1 was significantly higher than after placebo admini
stration. However, the inhaled beta(2)-agonist Salbutamol did not infl
uence submaximal and maximal (V) over dot(O2), f(c), [La-](b) or time
to exhaustion in the elite, nonasthmatic crosscountry skiers we studie
d. Thus, these results did not demonstrate any ergogenic effect of the
beta(2)-agonist used.