We report a case of acquired neuromyotonia in a patient with Staphyloc
occus aureus septicemia and a spinal epidural abscess. Autoantibodies
to voltage-gated potassium channels, which are associated with acquire
d neuromyotonia, were present during the patient's acute illness but b
ecame undetectable on clinical recovery. The spinal epidural abscess m
ay have triggered the production of these specific autoantibodies, res
ulting in clinically and electromyographically detectable neuromyotoni
a. (C) 1998 John Wiley & Sons, Inc.