Problems with meat quality are usually caused by aberrations in the bi
ochemistry and morphology of individual muscles, as well as by postmor
tem events. Poultry, like pigs, have been subjected to intense genetic
selection for rapid lean muscle growth. The selection traits are most
often associated with economic importance rather than biophysical sig
nificance which often results in stress syndromes (e.g. porcine stress
syndrome) and pale, soft, exudative (PSE) muscle conditions. The occu
rrence of PSE muscle and subsequent alterations in meat quality has be
en shown to be related to increases in muscle size, stressful preslaug
hter handling conditions, and rate of onset of rigor mortis. Morpholog
ical studies have revealed significant increases in fiber size, in add
ition to structural irregularities in PSE muscle. These structural irr
egularities include decreased capillary density, hypercontracted (gian
t) fibers, and myoplasmic calcium loading. The common theme between po
ultry and swine in the development of PSE muscle is predominately in a
naerobic fast-twitch muscles with low energy lactate metabolism yet ac
celerated onset of rigor mortis.