For better understanding of the etiology of ''post-supracondylar fract
ure cubitus varus,'' an in vitro anatomic experiment was performed. El
bow models were precisely photographed in 256 combinations of 10-degre
es increments of varus angulation, posterior angulation, internal rota
tion, and/or flexion contracture. Varus angulation was the most import
ant single factor contributing to deformity. Addition of flexion contr
acture or posterior angulation to a given varus angulation decreased a
pparent deformity, whereas addition of internal rotation worsened the
deformity. Control of varus angulation in the clinical setting, by wha
tever method, should minimize post-supracondylar fracture cubitus varu
s.