A possible agent for human non-A-E hepatitis has been identified and n
amed hepatitis G virus (HGV). HGV is also a flavivirus, and the clinic
al characteristics and risk factors of HGV infection may be similar to
those of hepatitis C virus infection. Hepatitis C virus infection may
manifest as a primary glomerulonephritis. The aim of this study is to
evaluate the prevalence of serum HGV RNA in 98 adult patients with bi
opsy-proven glomerular diseases. We found that only 3 patients (3%) ou
t of 98 with primary glomerulonephritis had HGV RNA. One of 59 (1.7%)
healthy controls was serum HGV RNA positive (p > 0.05). The prevalence
of serum HGV RNA was low in the patients with primary glomerulonephri
tis.