Je. Tetzlaff et al., CAUDA-EQUINA SYNDROME AFTER SPINAL-ANESTHESIA IN A PATIENT WITH SEVERE VASCULAR-DISEASE, Canadian journal of anaesthesia, 45(7), 1998, pp. 667-669
purpose: Spinal anaesthesia is selected for many lower extremity surgi
cal procedures each year in the United States with a high degree of sa
fety and efficacy. Even when adverse neurological outcomes have occurr
ed, anatomical abnormality or coagulopathy have been implicated in the
majority of cases. Epinephrine is used in high concentrations in many
of these anaesthetics to increase the duration and intensity of the b
lock. Although epinephrine is known to decrease spinal cord blood flow
, its use in normal patients has not caused complications. We report a
case where spinal anaesthesia with bupivacaine and epinephrine result
ed in anterior spinal artery compromise and the development of a cauda
equina syndrome postoperatively. Clinical Features: A 57-yr-old man w
ith severe coronary artery and peripheral vascular disease was schedul
ed for incision and drain of an abscess of the left thigh. He received
an atraumatic dural puncture and injection of 12.5 mg bupivacaine wit
h 0.2 ml 1:1000 epinephrine. During onset, he experienced a severe, pa
inful sensation of the thighs which resolved with development of the b
lock . Postoperatively, he was noted to have exacerbation of proximal
muscle weakness and decreased perineal sensation and rectal tone. Subs
equent EMG studies demonstrated proximal neuron loss consistent with c
auda equina syndrome, presumed to be related to insufficient of the an
terior spinal artery. Conclusion: Routine use of epinephrine in spinal
anaesthesia for patients with multi-organ vascular disease should be
considered carefully because of the possibility of vascular insufficie
ncy of the spinal cord which would be exaggerated by the vasoconstrict
ive effect of epinephrine.