Lipid oxidation is one of the main factors limiting the quality and ac
ceptability of meats and meat products. Oxidative damage to lipids occ
urs in the living animal because of an imbalance between the productio
n of reactive oxygen species and the animal's defence mechanisms. This
may be brought about by a high intake of oxidized lipids or polyunsat
urated fatty acids, or a low intake of nutrients involved in the antio
xidant defence system. Damage to lipids may be accentuated in the imme
diate past-slaughter period and, in particular, during handling, proce
ssing, storage and cooking. In recent years, pressure to reduce artifi
cial additive use in foods has led to attempts to increase meat stabil
ity by dietary strategies. These include supplementation of animal die
ts with vitamin E, ascorbic acid, or carotenoids, or withdrawal of tra
ce mineral supplements. Dietary vitamin E supplementation reduces lipi
d and myoglobin oxidation, and, in certain situations, drip losses in
meats. However, vitamin C supplementation appears to have little, if a
ny, beneficial effects on meat stability. The effect of feeding higher
levels of carotenoids on meat stability requires further study. Some
studies have demonstrated that reducing the iron and copper content of
feeds improves meat stability. Post-slaughter carnosine addition may
be an effective means of improving lipid stability in processed meats,
perhaps in combination with dietary vitamin E supplementation. (C) 19
98 Published by Elsevier Science Ltd. All rights reserved.