A patient is described who exhibited cerebellar ataxia after drinking
small amounts of alcohol. Intake of 5 g alcohol induced a gaze evoked
nystagmus, a scanning speech, a body sway after eye closure, and bilat
eral postural leg tremor. Kinematic and EMG analysis of fast wrist mov
ements showed normal movements before and marked hypermetria after alc
ohol intake. Dysmetria was due to abnormal programming of antagonist m
uscle activity.