To determine the relation between developmental dysphasia and EEG anom
alies during sleep, we compared 52 subjects with dysphasia with a cont
rol group of 20 children by using the ambulatory EEG method. Whereas 5
0% of the children with dysphasia experienced paroxysmal activity (PA)
, only two of the control group did. It is likely that paroxysmal abno
rmalities and language impairment are related to architectural dysplas
ia and neuron-migration disturbances. PA is frequent in subjects with
receptive developmental dysphasia and may be the cause of language det
erioration. When the occurrence of paroxysmal abnormalities during sle
ep is higher than 8% of total sleep time, we suggest the use of antiep
ileptic drugs.