Sleep apnea syndrome (SAS) is characterized by recurrent hypoxia durin
g sleep, excessive daytime sleepiness, snoring, abnormalities in sleep
architecture and deficits in daytime cognitive abilities. Numerous st
udies suggest that persons with SAS display a cognitive impairment pro
voked by the intermittent hypoxia produced during apneic episode. Alth
ough results are inconclusive, treatment of SAS with continuous positi
ve airway pressure (CPAP) appears to ameliorate significantly most of
neuropsychological deficits in apneic patients. On the other hand, SAS
in children may also cause a slowed neurological development, as well
as impairments in cognitive and behavioural functioning, including de
creased attention span, intermittent hyperarctivity, poor academic per
formance and developmental delay. However, the evidence is confined to
a small number of studies.