Sphincter tears: Vaginal delivery can lead to tears in the anal sphinc
ters. Total perineal distension following expulsion concerns less than
1% of all deliveries. Initially, sphincter tears generally go unnotic
ed although echographically detectable defects can be found in one-thi
rd of all primiparturients. The inner or outer sphincter may be involv
ed alone or in combination as is seen in half of the cases. Neurologic
al lesions: Moderate signs of incontinence (gas, urge) are frequently
reversible although the long-term outcome remains unknown. In half of
the cases, perineal denervation is secondary to stretch lesions of the
pudendal nerve terminasions. Favoring factors: Primiparity, forceps d
elivery, fetal macrosomy, and certain presentations (breech, occipitop
osterior) may favor sphincter lesions. Diagnosis: A complete examinati
on of the posterior perineum is required with anorectal manometry, a p
erineal electrophysiologic study, and a transanal ultrasound study whe
never function signs are found at the post partum follow-up. Treatment
: The therapeutic strategy is guided by the exploration results. In ca
se of symptomatic rupture of the external sphincter, sphicteroplasty i
s needed followed by functional rehabilitation therapy with biofeedbac
k. Women who have suffered traumatic lesions of the posterior perineum
should be carefully followed for signs of secondary incontinence. Ces
arean section may be indicated as a preventive measure in case of a ne
w pregnancy.