P. Delince et al., DIFFERENT ASPECTS OF THE CYCLOPS LESION FOLLOWING ANTERIOR CRUCIATE LIGAMENT RECONSTRUCTION - A MULTIFACTORIAL ETIOPATHOGENESIS, Arthroscopy, 14(8), 1998, pp. 869-876
After anterior cruciate ligament (ACL) reconstruction using a patellar
tendon autograft, 65 patients underwent second-look arthroscopy in con
junction with hardware removal. In 23 patients, hypertrophic tissue wa
s found in the anterior part of the knee. This tissue presented differ
ent aspects, from a well-synovialized nodule to a more disorganized fi
brous tissue according to patients' complaints. Endoscopic resection o
f this offending tissue was generally sufficient to obtain a satisfact
ory result. In patients presenting a loss of extension, the notch freq
uently had to be enlarged. We have found a multifactorial pathogenesis
to be likely: the nodule is a natural fibroproliferative tissue proce
ss originating either from drilling debris from the tibial tunnel or f
rom remnants of the ACL stump and, more rarely, from broken graft fibe
rs. Sometimes, when the graft is malpositioned, the scar tissue can re
sult from repeated graft impingement on the notch at terminal extensio
n. Formation of this aberrant tissue should be prevented by proper pos
itioning of the graft, by enlargement of the narrowed notch in chronic
cases, by using drills of increasing diameters to avoid production of
osteocartilaginous fragments, by meticulous resection of all drilling
debris and ACL remnants around the tibial tunnel, and by enlarging th
e notch roof if any contact with the graft is present when the knee is
fully extended intraoperatively.