Low back pain and lifestyle. Part II - Obesity - Information from a population-based sample of 29,424 twin subjects

Citation
C. Leboeuf-yde et al., Low back pain and lifestyle. Part II - Obesity - Information from a population-based sample of 29,424 twin subjects, SPINE, 24(8), 1999, pp. 779-783
Citations number
30
Categorie Soggetti
Neurology
Journal title
SPINE
ISSN journal
03622436 → ACNP
Volume
24
Issue
8
Year of publication
1999
Pages
779 - 783
Database
ISI
SICI code
0362-2436(19990415)24:8<779:LBPALP>2.0.ZU;2-0
Abstract
Study Design. A cross-sectional postal survey of 29,424 twin subjects aged 12-41 years obtained from a population-generated panel. Objectives. To determine whether obesity is associated with low back pain. Summary of Background Data. Despite a large number of epidemiologic studies in this area, it is unclear whether obesity and low back pain are positive ly associated, and if so, whether there is a causal association. Methods. The association and dose-response connection between body mass ind ex and nonspecific low back pain experienced by subjects in the preceding y ear were studied. Possible modifying effects of age, gender, type of work, and smoking were investigated. The prevalence of nonspecific low back pain was also studied in monozygotic twin pairs who were dissimilar in body mass index. Results. There was a modest positive association between body mass index an d low back pain that increased :;:with the duration of low back pain. The u nderweight subjects consistently reported lower prevalence of low back pain (odds ratios <1) than did those higher in weight. The dose-response curve was usually A-shaped. A positive monotonic dose response was apparent mainl y in those with long-lasting or recurrent low back pain. The positive assoc iation between body mass index and low back pain disappeared when monozygot ic twins who were dissimilar in body weight classification were studied. Conclusions. Obesity is modestly positively associated with low back pain, in particular with chronic or recurrent low back pain. However, because the association is weak, because there is no consistent positive monotonic dos e response, and because the link disappears in monozygotic twins who are di ssimilar in body mass index, it is unlikely that this association is causal . It is possible, however, that obesity plays a part in the chronicity of s imple low back pain. Therefore, those with recurring or long-term low back pain deserve further attention.