Epidemiology, clinical presentation and pathology of vasospasm

Citation
F. Turjman et al., Epidemiology, clinical presentation and pathology of vasospasm, J NEURORAD, 26, 1999, pp. 1S10-1S16
Citations number
41
Categorie Soggetti
Radiology ,Nuclear Medicine & Imaging
Journal title
JOURNAL OF NEURORADIOLOGY
ISSN journal
01509861 → ACNP
Volume
26
Year of publication
1999
Supplement
1
Pages
1S10 - 1S16
Database
ISI
SICI code
0150-9861(199903)26:<1S10:ECPAPO>2.0.ZU;2-D
Abstract
Symptomatic vasospasme or delayed cerebral ischemia associated with arterio graphic evidence of arterial constriction is currently the most important c ause of morbidity after acute subarachnoid hemorrhage. Symptomatic vasospas m usually develops between 4 and 12 days after subarachnoid hemorrhage. The re is typically a gradual deterioration of the level of consciousness accom panied by focal neurological deficits. 30% of patients who survived aneurys mal SAH develop delayed cerebral ischemia secondary to vasospasm. Vasospasm produces cerebral ischemia and infarction by hemodynamic mechanisms. Vasos pasm is an important independant predictor of poor outcome after aneurysmal SAH. Other conditions than aneurysmal subarachnoid hemorrhage such as trauma, tu mors, unruptured aneurysms, meningitis and ruptured AVM may be associated w ith vasospasm.