When the fatigue life Ni of a specimen of 10 mm in thickness is longer than
10(8) cycles, the average fatigue crack growth rate is much less than the
lattice spacing (similar to 0.1 Angstrom or 0.01 nm) that is 10(-11) to 10(
-12)m/cycle. In the early stage of the fatigue process, the crack growth ra
te should be much less than the average growth rate, and accordingly we can
not assume that crack growth occurs cycle by cycle.
In this paper, possible mechanisms for extremely high cycle fatigue are dis
cussed. Of some possible mechanisms, a special focus was put on a newly fou
nd particular fatigue fracture morphology in the vicinity of the fracture o
rigin (non-metallic inclusions) of a heat-treated alloy steel, SCM435, whic
h was tested to N greater than or equal to 108. The particular morphology o
bserved by SEM and AFM was presumed to be influenced by the hydrogen around
inclusions. The predictions of the fatigue limit by the root area paramete
r model are similar to 10% unconservative for a fatigue life of N-f = simil
ar to 10(8), though it successfully predicts the conventional fatigue limit
defined for N = 10(7). Thus, the fatigue failure for N greater than or equ
al to 10(8) is presumed to be caused by a mechanism which induces breaking
or releasing of the fatigue crack closure phenomenon in small cracks.
In the vicinity of a non-metallic inclusion at the fracture origin, a dark
area was always observed inside the fish-eye mark for those specimens with
a long fatigue life. Specimens with a short fatigue life of N-f = similar t
o 10(5) do not have such a dark area in the fish-eye mark. SEM and AFM obse
rvations revealed that the dark area has a rough surface quite different fr
om the usual fatigue fracture surface in a martensite lath structure.
Considering the high sensitivity of high-strength steels to a hydrogen envi
ronment and the high hydrogen content around inclusions, it may be conclude
d that the extremely high cycle fatigue failure of high-strength steels fro
m non-metallic inclusions is caused by environmental effects, e.g. hydrogen
embrittlement coupled with fatigue.