We evaluated ciliochoroidal effusion (CE) by ultrasound biomicroscopy (UBM)
following diode endophotocoagulation at the end of the vitreoretinal surge
ry. The aim of our study was to assess any differences in the CE morphology
following diode endophotocoagulation or transpupillary krypton photocoagul
ation, and to demonstrate the influence of diabetes and intravitreal surger
y on CE formation. Sixty-six consecutive patients were divided in to four g
roups. Twenty-nine patients with proliferative retinopathy underwent transp
upillary krypton photocoagulation; 11 diabetic patients underwent vitreoret
inal surgery and diode endophotocoagulation; 18 nondiabetics underwent vitr
eoretinal surgery and diode endophotocoagulation; 8 consecutive nondiabetic
patients were the control group and underwent vitreoretinal surgery, witho
ut laser treatment. UBM was performed in the four groups before and after l
aser treatment, if performed. We determined, by UBM, not only the presence,
but also the thickness of CE. CE was present in all the patients treated b
y laser, diabetics and nondiabetics, and its thickness was not correlated w
ith the number of laser spots (p = 0.28). CE was seen ultrasonically in all
the patients undergoing transpupillary photocoagulation or endophotocoagul
ation, regardless of diabetes and surgical trauma.