Nine babies with pure oesophageal atresia were treated in our institution i
n the years 1979-1996. All received a feeding gastrostomy as their initial
operation. After initiation of gastrostomy feeds seven (78%) developed gast
ric complications, including two posterior gastric perforations tone fatal)
. We propose that the high complication rate is due to a small, abnormal st
omach that is vulnerable to damage by operative trauma and the effects of h
andling large volumes of feed. We hypothesise that the stomach is abnormal
because it has not been exposed to the maturing effects of amniotic fluid i
n utero. Feeds should be introduced very cautiously to these babies and bui
lt up very slowly.