Plantar heel pain syndrome has been attributed to entrapment neuropathy, pl
antar fasciitis, calcaneal spurs, and stress fractures of the calcaneus, Al
though deteriorated mechanical properties of the heel pads may play an impo
rtant role in the pathogenesis of heel pain syndrome, this has received lit
tle notice, In this study, a specially designed compression relaxation devi
ce with a push-pull scale and a 10-MHz linear array transducer was used to
determine thickness of the heel pad under different loading conditions, Twe
nty consecutive patients aged 29 to 77 years with unilateral plantar heel p
ain syndrome were enrolled. Thickness of heel pad bilaterally was measured
when the heel pad was compressed by serial increments of 0.5 kg to a maximu
m of 3 kg and then relaxed sequentially, The load-displacement curve during
a loading-unloading cycle was plotted, and the compressibility index and e
nergy dissipation ratio of the heel pad were calculated accordingly, Phase
I displacement of the heel pad (from 0 to 1 kg load) on the painless side w
as greater than that on the painful side (P < 0.01), but there was no stati
stically significant difference between painless and painful sides in thick
ness of unloaded heel pads, compressibility index, or energy dissipation ra
tio (P > 0.05), In conclusion, the affected heel pad in plantar heel pain s
yndrome was stiffer under light pressure than the heel pad on the painless
side. The changed nature of chambered adipose tissue in a painful heel pad
may be responsible for its increased stiffness under light pressure.